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Dhia Hamed: CONTRIBUTION TO THE GEOPHYSICAL STUDY OF A DEEP WATER TABLES IN THE SOUTH GABES SOUTH OF TUNISIA ; 140-19 Booth 69 Jain Ratanchand * , Sinha Amrendrakumar: HYDROGEOLOGICAL FRAMEWORK OF THE OVER-EXPLOITED MULTI AQUIFER SYSTEM IN NORTH GUJARAT, INDIA PROBLEMS AND PROSPECTS OF SUSTAINABILITY 140-20 Booth 70 Leticia Flores-Marquez * , Gabriel Jimenez-Suarez, Rene Chavez-Segura, Raymundo Martinez-Serrano: 3D MODEL OF THE AQUIFER DYNAMIC EVOLUTION OF THE VALLE DE PUEBLA AQUIFER SYSTEM, MXICO 140-21 Booth 71 Beretta Giovanni Pietro, Cambareri Maria Nunzia, Masetti Marco * , Pilati Marco, Riparbelli Carlo: GROUNDWATER PROTECTION AND MANAGEMENT OF POROUS ACQUIFERS IN LOMBARDY PLAIN ITALY ; 140-22 Booth 72 Pappalardo Giovanna * , Ferrara Vincenzo, Maugeri Salvatore: AQUIFER VULNERABILITY EVALUATION THROUGH DIFFERENT METHODOLOGIES IN AN AREA OF CENTRAL-EASTERN SICILY 140-23 Booth 73 Stojiljkovic Dragica * , Rajic Milorad: HYDRO CHEMICAL ZONING OF GROUNDWATER OF VOJVODINA 140-24 Booth 74 Perepechko Yuri * , Dorovsky Vitaly: NON-LINEAR DYNAMICS OF SATURATED POROUS MEDIA 140-25 Booth 75 Abukova Leyla * : THE DESCENDING GEOMIGRATION AS THE FACTOR OF ACCUMULATION OF OIL AND GAS IN BOTTOMS SEDIMENTAL OILBURIED BASIN 140-26 Booth 76 De Luca Domenico * , Lasagna Manuela: USE OF THE DILUTION ATTENUATION FACTOR IN THE GROUNDWATER CONTAMINATION EVALUATION, WITH PARTICULAR REFERENCE TO NITRATE POLLUTION 140-27 Booth 77 Hwang Seho * , Shin Jehyun, Kim Jeon-Il, Ji Sejeong, Park Kwon-Gyu, Song Young-Soo, Lee Sang-Kyu: ASSESSMENT OF SEAWATER INTRUSION USING GEOPHYSICAL AND HYDROGEOCHEMICAL SURVEY IN YEONGGWANG-GUN, KOREA 140-28 Booth 78 Jackowicz-Korczynski Jacek * : UTILISATION OF WATERS FROM CLOSED MINES 140-29 Booth 79 Jimenez-Espinosa Rosario, Jimenez-Millan Juan * , Diaz-Beltran Leticia, Vazquez Mercedes, Abad Isabel: REACTIVE MINERAL ASSEMBLAGES OF THE ALLUVIAL AQUIFER MATRIX OF THE GUADALQUIVIR RIVER NORTHERN JAEN PROVINCE, SPAIN ; . CONTROL ON THE GROUNDWATER QUALITY 140-30 Booth 80 Kumar Sunil * , Saini D. S.: GROUNDWATER CONTAMINATION IN AMRITSAR CITY, PUNJAB, INDIA 140-31 Booth 81 Lasagna Manuela * , De Luca Domenico Antonio: VERTICAL HYDROCHEMICAL DIFFERENTIATION AND NITRATES DISTRIBUTION IN THE SUPERFICIAL AQUIFER OF THE EASTERN SECTOR OF PIEMONTE PLAIN 140-32 Booth 82 Dokmanovic Petar * : GROUNDWATER RESOURCES OF NEOGENE ARTESIAN BASINS IN SERBIA-SOUTH FROM SAVA AND DANUBE 140-33 Booth 83 Jata Idriz * , Kavaja Vladimir, Papaqako Mimoza: THE ASSESSMENT OF THE AQUIFERS AT SHKODRA REGION, ALBANIA 140-34 Booth 84 Khalil Mohamed, Knoblich Klaus, Hassaneen Abdelrady, Ragab Said * , Sherief Salah: GEOELECTRIC INVESTIGATION OF THE SALINITY PROBLEM IN EL-ARISH AREA, NORTHERN SINAI, EGYPT 140-35 Booth 85 Barazzuoli Piero, Mocenni Benedetta * , Nocchi Monica, Rigati Roberto, Salleolini Massimo: HYDROGEOLOGICAL STUDY OF THE FOLLONICA-SCARLINO PLAIN SOUTHERN TUSCANY, ITALY.
P&U had also repeatedly attracted criticism for misleading advertising, notably in the USA. Between 1998 and 2000, P&U received five warning letters from the US Food & Drug Administration. In the two most recent, the company was taken to task for making headline claims about the "selectively" of its product - suggesting that it acted on the bladder, with less dryingup effects in the mouth. P&U evidence was based not on clinical trials, but on studies with cats. In the UK, the company resisted regulatory pressure for over a year. The Patients Association became aware of this too late: they had already supported the TV campaign and accepted a large donation from P&U. In turn, they said a nice thank-you to the company and gave it one of their `Platinum Awards'.xlvi DG Enterprise has yet to address the problems that may arise from this. One is that Pharma companies get `two bites of the cherry' pleading their own case and then having their views 'represented' in consultations and regulatory matters by more or less captive patient representatives and organisations. Another is the vulnerability of patient interests to overtures from companies and trade associations. Many patient groups struggle for survival and DTC advertising campaigns can empower them. Directly and indirectly, such campaigns draw attention to the needs of special and deserving interests and the disadvantages they face. For the groups that represent them, DTCA can bring in more members, perhaps more column-inches and sound bites, greater exposure and prominence and sense of achievement, and sometimes large grants from companies too. The G10 has acknowledged the problem exists, to the extent of proposing that "the Commission should provide core funding for European Patient groups to enable them to participate independently in the debate and decision-making on health matters at a European level". But how much would it help if funding were steered by DG Enterprise, when its primary commitment is to entrepreneurship and market growth - and given its perspectives on the relationship between trade and health? The G10 Task Force the majority ; xlvii seems to take it very much for granted that DTC marketing can provide patients with information they want, need and would benefit from. People would become better informed, disease awareness would grow and the stigma of illness would be assuaged. Moreover, trade liberalisation would encourage companies to innovate the important new medicines that people want now. Simple: win win and diazepam.
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| Cyclobenzapr flexeril medication cyclobenzaprinePlatelet-activating factor to platelets of different animal species; Eur. J. Pharmacol. 105 309315 Jamaluddin M 1991 New perspectives in blood platelet aggregation; Curr. Sci. 61 527533 Jamaluddin M and Krishnan L K 1990 Adenosine and ATP: mutually exclusive modifiers of the kinetics of ADP-induced aggregation of calf-platelets; J. Biosci. 15 389396 Jamaluddin M and Krishnan L K 1987 A spectrophotometric method for following initial rate kinetics of blood platelet aggregation; J. Biochem. Biophys. Methods 14 191200 Jamaluddin M, Krishnan L K and Thomas A 1988 Ajoene inhibition of platelet aggregation: Possible mediation by a hemoprotein; Biochem. Biophys. Res. Commun. 153 479486 Janero D R, Burghardt B and Burghardt C 1988 Specific binding of platelet-activating factor ; to the intact canine platelet; Thromb. Res. 50 789802 Kloprogge E and Akkerman J W N 1984 Binding kinetics of PAF-acether ; to intact human platelets; Biochem. J. 223 901909 Kromhaut D, Bosschieter E B and Coulander C D L 1985 The inverse relation between fish consumption and 20-year mortality from coronary heart disease; N. Engl. J. Med. 312 12051209 Kudolo G B and Harper J K 1990 Estimation of platelet-activating factor receptors in the endometrium of the pregnant rabbit: regulation of ligand availability and catabolism by bovine serum albumin; Biol. Reprod. 43 368377 Lapetina E G and Siegal F L 1983 Shape-change induced in human platelets by platelet-activating factor. Correlation with the formation of phosphatidic acid and phosphorylation of a 40, 000-dalton protein; J. Biol. Chem. 258 72417244 McCullock R K and Vandongen R 1990 Mechanisms of platelet-activating factor-induced aggregation and secretion in human platelets; Prostaglandins 39 1321 Moon D G, Vander Zee H, Morloin K D, Krasdomski J A, Kaplan J E and Fenton J W II 1990 Platelet activating factor and sheep platelets: a sensitive new bioassy; Thromb. Res. 57 551564 Moore J B Jr, Fuller B L, Falotico R and Tolman E 11985 Inhibition of rabit platelet phosphodiesterase activity and aggregtion by calcium channel blockers; Thromb. Res. 40 401411 Namm D H, Tadepalli A S and High J A 1982 Species specificity of the platelet responses to Thromb. Res. 25 341350 Ng D S and Wong K 1988 Effect of sulphydryl reagents on PAF binding to human neutrophils and platelets; Eur. J. Pharmacol. 154 4752 Ostermann G, Hofmann B, Kertscher H P and Till V 1990 PAF-agonistic and antagonistic behaviour of new synthetic ether phospholipids. I. Studies on blood platelets in vitro; J. Lipid Mediators 2 2131 Prescott S M, Zimmerman G A and McIntyre T M 1990 Platelet-activating factor; J. Biol. Chem. 265 1738117384 Santoro S A and Lawing W J Jr 1989 Modulation of an RGDS binding site on the platelet membrane glycoprotein IIb-IIIa complex; Biochem. Biophys. Res. Commun. 160 189195 Selak M A and Smith J B 1989 Platelet activating factor-induced calcium mobilization in human platelets and neutrophils: effects of PAF-acether antagonists; J. Lipid Mediators 1 125137 Shekelle R B, Missel L, Paul 0, Schryock A M and Stamler J 1985 Fish consumption and mortality from coronary heart disease; N. Engl. J. Med. 313 820 Shukla S D and Hanahan D J 1982 AGEPC platelet activating factor ; induced stimulation of rabbit platelets: effects on phosphatidylinositol, di- and tri-phosphoinositides and phosphatidic acid metabolism; Biochem. Biophys. Res. Commun. 106 697703 Seiss W 1989 Molecular mechanisms of platelet activation; Physiol. Rev. 69 58178 Sreedevi C, Thomas A and Jamaluddin M 1989-Spectrophotometric platelet aggregation assay to measure single platefet disappearance: An evidence; Curr. Sci. 56 483485 Suzuki Y, Miwa M and Harada M 1988 Release of acetylhydrolase from platelets on aggregation with platelet activating factor; Eur. J. Biochem. 172 117120 Swann P G, Parent C A, Croset M, Fonlupt P, Lagarde M, Venton D L and Le Breton G C 1990 Enrichment of platelet phospholipids with eicosa pentaenoic acid and docosa hexa enoic acid inhibits thromboxane A2 prostaglandin H2 receptor function; J. Biol. Chem. 265 2169221697 Tahrouli L, Floch S and Cavern I 1990 Functional validation of platelet-activating factor receptor sites characterized biochemically by a specific and reproducible [3H] platelet activating factor binding in human platelets; J. Pharmacol. Exp. Ther. 252 12211227 Thomas A, Krishnan L K and Jamaluddin M 1988 Aspirin an apparently co-operative modifier of arachidonate and A23187-induced aggregation of gel-filtered calf platelets; 57th Annual meeting of the Society of Biological Chemists, India, Abstract III Tokumura A, Yoshida J, Okasaka N, Fukuzawa K and Tsukantani H 1987 Platelet aggregation induced and elocon.
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W.F. PEATE, M.D., M.P.H., is an associate professor of family and community medicine at the University of Arizona College of Medicine, Tucson, and is an associate professor at the university's College of Public Health. He received his medical degree from Dartmouth Medical School, Hanover, N.H., and his master's degree in public health at Harvard University, Boston, Mass. He completed an occupational medicine residency at the University of Arizona College of Medicine. Address correspondence to W.F. Peate, M.D., M.P.H, University of Arizona College of Public Health, 1295 N. Martin Ave., P.O. Box 245210, Tucson, AZ 85724-5210 e-mail: peate email.arizona ; . Reprints are not available from the author. Author disclosure: Nothing to disclose. rEFErEncEs and flomax.
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Figure 15.16 Model of graded neural injury that combines 109 110 Seddon and Sunderland clinicalpathologic classifications. Microanatomic changes in cranial nerve injury are demonstrated in cross-section after Terzis and 201 Smith ; . The potential for appropriate axonal regeneration across the site of injury is dictated principally by the status of connective tissue elements. during this period. Routine electrophysiologic tests therefore fail to detect nerve conduction as it occurs, thereby delaying differentiation of neuropraxia from degeneration. Nerve excitability testing: Minimal excitability testing 112 with the Hilger nerve stimulator has provided a readily accessible method of facial nerve assessment. The test is indexed according to threshold for visually detectable activity generated by surface stimulation of a facial nerve branch. The test reflects elevated thresholds for neuromuscular stimulation produced by axonal disruption and degeneration. The lowest stimulus intensity that consistently excites all branches on the uninvolved 113 side establishes the normal threshold. A 23.5 mA difference between the uninvolved and involved sides is reported to suggest impending denervation. This test offers technical advantages in the portability of the necessary equipment and the use of minimal stimulation which is more comfortable for the patient than maximal stimulation tests. The test, however, introduces subjectivity in that it relies on visual detection of a response of a limited number of facial muscles. In addition, current levels at threshold for peripheral branches are likely to selectively activate large nerve fibers with lower thresholds ; and those fibers closer to 115 the stimulating electrode, thereby excluding an unknown proportion of motor fibers from the assessment!
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Courses e.g., acute gouty arthritis ; and implied that chronic use is not recommended. Therefore, we chose to target chronic users of indomethacin, defined as more than 1 pharmacy claim for indomethacin during each intervention quarter. R e g rding the anticholinergic agents disopyramide, cyclobenzaprine, dicyclomine, and methocarbamol, it is generally felt that these agents should be avoided in older adults due to their potential adverse effects.2 We also decided to target disopyramide based on our pharmacy utilization and the choices of alternate antiarrhythmic agents. We added dicyclomine and hyoscyamine to our list of targeted medications based on pharmacy utilization and the individual agent's potential for adverse effects in this patient population. We decided to no longer target propoxyphene due to decreased pharmacy utilization of this agent. Since methyldopa was listed in the Beers criteria, has the potential to cause bradycardia and exacerbate depression in older adults, and was highly utilized in our patient population, we chose to include it as one of our targeted medications. Additionally, there are many alternate treatments for hypertension that have less serious side effects. We calculated quarterly measurements of the proportion of older adult members who filled a prescription for a contraindicated drug. We also calculated the proportion of older adult members who filled a prescription for 1 or more of the subset of 7 targeted contraindicated drugs that was consistent throughout the entire 4-year intervention period. This intervention program with prescribers of target contraindicated drugs for older adult members in this health plan is ongoing. The base period for assessing the impact of the intervention program was the fourth calendar quarter Q4 ; of 1999. The measurement period for this study included 16 calendar quarters from 2001 Q1 through the 2003 Q4. Letters were mailed to prescribers of medications contraindicated in older patients after the end of each calendar quarter. The letter described the program and included a provider pro f i l Appendix ; . In some cases, the prescriber was not the primary care provider PCP ; for the particular patient the p rescriber may have been another PCP or a specialist ; . There f o re, each prescriber and PCP received a profile containing their names, the full name and HMO identification number of each patient who received contraindicated drugs in that specific quarter, the generic name of the contraindicated drug s ; that each patient received in that quarter as well as the reason for the contraindication, any formulary alternatives if available ; , and identification of the physician prescribing each drug. In addition to sending a prescriber-specific profile, a clinical pharmacist contacted some prescribers by telephone. Each calendar quarter, prescribers with the highest volume of patients receiving the target contraindicated drugs were contacted by telephone. A clinical pharmacist called prescribers to discuss the target patients' drug use and treatment plan. Alternative therapeutic options were generally discussed with.
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For health advice, the guide directs the public to one of four sources of help: calling NHS Direct to speak to a nurse; using NHS Direct Online; using the NHS Direct Healthcare Guide; and asking a local pharmacist. According to the guide, NHS Direct Online provides information about a variety of conditions and treatment choices. But a search of the website reveals little information about prescription medicines.213 The NHS Direct Healthcare Guide provides information on non-prescription medicines only and with the exception of a few examples is non-product specific. The local pharmacist is presented as a source of advice about minor illnesses only. Patients have the right to complain about any aspect of NHS care including decisions not to provide services or certain treatments. However, in a cash-strapped NHS where the principles of collective responsibility and social solidarity reign, the chances of getting decisions not to provide certain treatments overturned are small. In most cases, if patients can't get the treatments they want from the NHS, they have little option but to opt out and seek that treatment privately.
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Ore than 5 million diagnostic and interventional cardiac catheterizations are performed each year in the United States.1 Cardiac catheterization is considered the gold standard for the diagnosis, evaluation, and treatment of cardiac diseases. Although it has reduced morbidity and mortality for cardiovascular disease, this invasive procedure is not free of complications.2 From June 2004 through December 2006, PA-PSRS received more than 1, 400 reports referencing cardiac catheterization procedures. Almost half of the reports were classified as complication of the procedure. More than 230 of these complications were vascular complications associated with the access site, including bleeding, hematoma formation, retroperitoneal bleed, pseudoaneurysm, and arteriovenous fistula A-V ; formation. Of the 230 vascular complications reported, the most often stated causes of the problem were medication errors, inconsistencies in patient assessments, unrecognized changes in patient condition, sheath removal, and lack of appropriate interventions when complications occurred. This article presents strategies for reducing vascular complications associated with cardiac catheterizations through identification of associated risk factors and implementation of risk reduction strategies. Both cardiologists and nurses play a vital role in the early recognition and management of these complications. For example, the following report submitted to PA-PSRS demonstrates a missed opportunity to recognize signs and symptoms of bleeding at the access site, which led to serious vascular complications that required additional intervention in the operating room OR ; . Patient underwent a cardiac catheterization on Friday. The sheath was removed Saturday morning. Patient developed a large hematoma in the right groin area in the afternoon. Pressure applied to the site for 15 minutes. Sandbag applied for more than two hours. By evening, hemoglobin and hematocrit had dropped to 6.6 and 21.8. Patient had to return to the OR to stop the bleeding and evacuate the hematoma. Patient was transferred to CVU post-op.
Information technology or related areas. We may acquire, mortgage, liquidate or sell real estate and intellectual property rights in Switzerland or abroad. The duration of our company is unlimited. We may be dissolved at any time by a shareholders' resolution. In the case of a dissolution by way of liquidation, such resolution would require the approval of the majority of votes present at the shareholders' meeting. In the case of a dissolution without liquidation, at least two-thirds of the votes present at the meeting would have to vote their shares in favor of such resolution. Dissolution is also possible by adjudication of bankruptcy or by decision of a judge, if shareholders holding at least 10% of the registered share capital requested dissolution for valid reasons. Under Swiss law, any surplus arising out of a liquidation i.e., after the settlement of all claims of all creditors ; would be distributed to the shareholders in proportion to the paid-in nominal value of their shares. Shareholders may pass a resolution to merge with another corporation at any time. Such a resolution would require the consent of at least two-thirds of all votes present at the necessary shareholders' meeting. Disclosure of Principal Shareholders Under the Swiss Stock Exchange Act, holders of our voting shares would be required to notify us and the SWX of the level of their holdings whenever such holdings reach or exceed, or in some cases, fall short of, certain thresholds--5%, 10%, 20%, 331 and 662 3%--of our registered share capital, whether or not the shareholder has the right to cast votes based on the shares. Following receipt of such notification we would be required to inform the public by publishing the information in the Swiss Official Commercial Gazette and in at least one of the principal electronic media that disseminate stock exchange information. Mandatory Tender Offer Under the Swiss Stock Exchange Act, shareholders and groups of shareholders acting in concert who acquire more than 331 3% of the voting rights of Novartis shares would be required to submit a takeover bid to all remaining shareholders. This mandatory bid obligation may be waived by the Swiss Takeover Board or the Swiss Federal Banking Commission under certain circumstances, in particular if another shareholder owns a higher percentage of voting rights than the acquirer. If no waiver is granted, the mandatory takeover bid would have to be made pursuant to the procedural rules set forth in the Swiss Stock Exchange Act and the ordinances enacted thereunder. Board of Directors Pursuant to Swiss law, each member of our Board must hold at least one of our shares. Directors must retire when they reach age 71, although the General Meeting may grant an exemption from this rule. We have no mandatory retirement age for executive officers. American Depositary Shares We incorporate by reference the disclosure regarding our ADS program included in the registration statement on Form 20-F A File No. I-15024 ; , as filed with the Commission on May 9, 2000, in the section entitled ``Part II--Item 14. Description of Securities to be Registered--American Depositary Receipts.'' On May 3, 2001, we filed an Amendment No. 2 to the Amended and Restated Deposit Agreement, dated as of May 7, 2001, pursuant to the Registration Statement on Form F-6 File No. 333-13446 ; . The Amendment No. 2 changed the ADS-to-share ratio from 40-to-1 to 1-to-1. On January 31, 2002, we filed a Restricted Issuance Agreement dated as of January 11, 2002, supplementing Amendment No. 2 to the Amended and Restated Deposit Agreement dated as of May 3, 2001, as an exhibit to the Registration Statement on Form F-3 File No. 333-81862 ; . The Restricted Issuance Agreement supplemented the Deposit Agreement to permit the deposit of restricted ADSs into a parallel facility to the ADR facility established in the Deposit Agreement. 99, because is cyclobenzaprine a narcotic.
At the heart of ORBIS's sight-saving work is the world's only Flying Eye Hospital, a DC10 aircraft converted into a state-of-the-art operating room and teaching facility. On board is an international team of health professionals from more than ten different countries, working together to share sight-saving skills around the world. Additionally, more than 400 leading surgeons and medical volunteers donate their time to provide training and technical support for a wide range of blinding diseases and conditions. The unparalleled international cooperation demonstrated by the Flying Eye Hospital serves as an inspiration to health care providers and government leaders. Everywhere it travels, the plane's high profile, through front page media coverage, increases awareness of global blindness and builds local support for potential solutions. Through this unique medium, ORBIS is raising community consciousness regarding blindness prevention and the need for quality eye care. The mission of ORBIS actually began in 1972, when ORBIS founder Dr. David Paton, chairman of the Baylor College of Medicine's ophthalmology department, came up with the idea of a globally mobile eye care facility. He discussed with his friend Betsy Trippe DeVecchi the idea of outfitting an aircraft and Betsy sought the advice of her father, Pan American World Airways founder Juan Trippe, who invited Dr. Paton, Betsy and current ORBIS Chairman A.L. Ueltschi to New York City to discuss the idea. It was resolved to create ORBIS and in 1974 United Airlines agreed to donate the first ORBIS plane, a McDonnell Douglas DC-8. It took eight more years to pull together the necessary resources and convert the aircraft into the world's only Flying Eye Hospital, which finally took off from Houston on March 1, 1982 and flew to Panama to carry out the first ever ORBIS program. In 1994, the ORBIS aircraft was upgraded to the current DC-10, its purchase funded by Chairman and CEO of FlightSafety International A.L. Ueltschi, Hong Kong businessman Y.C. Ho and a third anonymous donor. Subsequently, its conversion into a fully-equipped.
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Monthly Garage Sale Great Bargains 1st Saturday of each month at the Hospice Warehouse, 252 Forrest Hill Road, Forrest Hill. 8am 12 noon. Ph Marlene Grigsby 410 3006 Tours of North Shore Hospice. Last Friday of each month, 10.15 11am. An opportunity to view the North Shore Hospice Unit and hear more about our services. Oxford Charitable Trust North Shore Hospice Golf Day Players who sign up will have a great day of golf, with lunch, dinner and all day drinks provided. Each player will also receive a wonderful gift pack, a Hospice Golf Tournament polo shirt and a great prize. $150 per player. Anyone interested in helping or in playing please Ph Joan or Ross Finlayson 489 9083 or Neil Crawford 415 6788 Annual Fun Tennis Tournament Join us for a great day of food, drink, fun. oh, and tennis at one of 30 private tennis courts on the North Shore and over the Bridge. Each player receives a wonderful gift, and the top male and female on each court takes home a prize. Entry Fee: Players $85.00 Non-Players $65.00. For enquiries contact Rosemary Hutcheson PH 486-1688 North Shore Hospice Bowling Tournament. Organised by Bowls North Harbour Inc. Bowlers who sign up will have a great day of bowling at the Browns Bay Bowling Club with lots of fun, lunch, raffles and auction. $100 per team any combination of fours. If you are interested in participating in this great day please contact Keith Gerrie 478 3011 Entertainment Book Launch The next Entertainment Book will be available from this date at North Shore Hospice reception. Do not miss out! Thousands of dollars worth of discounts for dining, events, travel, concerts etc. Ph Karen Anderson 486 1688 28 Apr Morning Tea with Lindsey Dawson Come along and hear this wonderful writer, speaker and word whiz, the founding editor of three national magazines, More, Next & Grace. She has now moved on to focus on her own projects & writing books. Tickets $20 each. 10am start. Check out lindseydawson Ph Jean Douglas 412 2266 Holiday Auction Brunch Come along for brunch and enjoy a great and entertaining auction for Winter Weekend Getaways. Great venues to bid for! Ph Sandra Turner 410 6810 7 August North Shore Hospice Annual Ladies 9 Hole Golf Day Come and enjoy the company of your friends and colleagues, play golf, have lunch and view the latest trends in fashion. Ph Jan Aickin 4787122 or 0274-291891 Movie or concert Evening This month we are planning another movie night or alternatively a concert with a difference. Ph Gill Hill 446 3375 Platinum Homes Tour In 2006, we will be visiting some spectacular homes in the northern East Coast Bays area from Rothesay Bay to Long Bay. Come along with your friends for a wonderful day! Ph Sandra Turner 4106810 2nd Annual Fashion Parade For The Fab 40s + ; Such was the success of the inaugural Fashion Parade for those "stepping out" over 40, that we are now planning the next one. Enjoy a night of fashion, inspiration, fun and laughter, delicious wine and cheese, spot prizes and a take-home goody bag. For enquiries contact Rosemary Hutcheson Ph 486 1688.
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For definition of Groups, see Preamble Evaluation. Supplement 7: 1987 ; p. 184 ; CAS No.: 4342-03-4 Chem. Abstr. Name: 1H-Imidazole-4-carboxamide, 5- 3, ; A. Evidence for carcinogenicity to humans inadequate ; No epidemiological study of dacarbazine as a single agent was available to the Working Group. Occasional case reports of exposure to dacarbazine, especially in the presence of concurrent therapy with other putative carcinogens, such as ionizing radiation, alkylating agents and other potent oncotherapeutic drugs, do not constitute evidence of carcinogenesis [ref: 1]. In a large systematic follow-up of patients with Hogdkin's disease treated with an intensive chemotherapeutic combination including dacarbazine plus adriamycin, vinblastine and bleomycin ; but no alkylating agent, preliminary evidence suggested no excess of acute nonlymphocytic leukaemia in the first decade after therapy [ref: 2]. B. Evidence for carcinogenicity to animals sufficient ; Following its oral or intraperitoneal administration to rats, dacarbazine produced tumours at various sites, including mammary gland, thymus, spleen and brain, in as little as 18 weeks after initial exposure [ref: 1]. After its intraperitoneal administration to rats at the end of pregnancy, dacarbazine produced tumours, the majority of which were malignant neurinomas, in offspring [ref: 3]. Dacarbazine produced tumours at various sites, including lung, haematopoietic tissue and uterus, after intraperitoneal administration to mice [ref: 1]. C. Other relevant data Dacarbazine did not induce sister chromatid exchanges in lymphocytes of treated patients in one study. It gave weakly positive results for induction of sister chromatid exchanges in Chinese hamster cells in vitro and was mutagenic to cultured rodent cells and to bacteria [ref: 4]. Overall evaluation Dacarbazine is possibly carcinogenic to humans Group 2B ; . For definition of the italicized terms, see Preamble Evaluation. Also see previous evaluation: Vol. 26 1981 ; References 1. IARC Monographs, 26, 203-212, 1981 Valagussa, P., Santoro, A., Bellani, F.F., Franchi, F., Banfi, A. & Bonadonna, G. 1982 ; Absence of treatment-induced second neoplasms after ABVD in Hodgkin's disease. Blood, 59, 488-494 3. Zeller, W.J. 1980 ; Prenatal carcinogenic action of 5- 3, 3-dimethyl-1-triazeno ; imidazole-4carboxamide DTIC ; in the offspring of BD IX rats Ger. ; . Arch. Geschwulstforsch., 50, 306-308.
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